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A Conserved uORF Regulates APOBEC3G Translation and Is Targeted by HIV-1 Vif Protein to Repress the Antiviral Factor
ArticleAbstract: The HIV-1 Vif protein is essential for viral fitness and pathogenicity. Vif decreases expression ofPalabras claves:APOBEC3G, HIV-1, mRNA, translation, UORF, VifAutores:Batisse J., Cabrera-Rodriguez R., Cimarelli A., Etienne L., Gilmer O., Libre C., Marquet R., Paillart J.C., Santiago Xavier Guerrero, Seissler T., Stupfler B., Valenzuela-Fernandez A., Verriez C., Weber M.M.Fuentes:googlescopusHiv-1 replication and the cellular eukaryotic translation apparatus
ReviewAbstract: Eukaryotic translation is a complex process composed of three main steps: initiation, elongation, anPalabras claves:Auxiliary proteins, Frameshift, HIV-1, IRES, Leaky-scanning, Ribosome shunting, translationAutores:Batisse J., Bernacchi S., Libre C., Marquet R., Paillart J.C., Santiago Xavier GuerreroFuentes:googlescopusTranslational regulation of APOBEC3G mRNA by Vif requires its 5′UTR and contributes to restoring HIV-1 infectivity
ArticleAbstract: The essential HIV-1 viral infectivity factor (Vif) allows productive infection of non-permissive celPalabras claves:Autores:Batisse J., Decoville T., Laumond G., Libre C., Marquet R., Mercenne G., Moog C., Paillart J.C., Richer D., Santiago Xavier GuerreroFuentes:googlescopus