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Adipokine pathways are altered in hippocampus of an experimental mouse model of Alzheimer’s disease
ArticleAbstract: A growing body of evidence suggests that β-amyloid peptides (Aβ) are unlikely to be the only factorPalabras claves:ALZHÉIMER, APP/PS1, hippocampus, leptin, prolactinAutores:Abad S., Antoni Camins, Artiach G., Folch J., Pallás M., Pedros I., Petrov D., Ramon-Duaso C., Sureda F.X., Zárate C.B.Fuentes:scopusEarly Preclinical Changes in Hippocampal CREB-Binding Protein Expression in a Mouse Model of Familial Alzheimer’s Disease
ArticleAbstract: The molecular basis of memory loss in Alzheimer’s disease (AD), the main cause of senile dementia, iPalabras claves:Alzheimer disease, APPswe/PS1dE9, CBP, CREB, hippocampusAutores:Abad S., Antoni Camins, Auladell C., Busquets O., Carro E., Casadesús G., Ettcheto M., Folch J., Olloquequi J., Pallás M., Pedros I., Petrov D., Sanchez-Lopez E., Zárate C.B.Fuentes:scopusEvaluation of the Role of JNK1 in the Hippocampus in an Experimental Model of Familial Alzheimer’s Disease
ArticleAbstract: c-Jun N-terminal kinases (JNKs), which belong to a mitogen-activated protein kinase (MAPK) family, aPalabras claves:Alzheimer’s disease, amyloid, Amyloid precursor protein, APPswe/PS1dE9, BACE1, JNK1, PGC-1α, Transgenic mouse models, β-secretase, βAAutores:Abad S., Antoni Camins, Auladell C., Ettcheto M., Folch J., Luque M., Pallás M., Pedros I., Petrov D., Verdaguer E.Fuentes:scopusReview of the advances in treatment for Alzheimer disease: Strategies for combating β-amyloid protein
OtherAbstract: Introduction Alzheimer disease (AD) is a major neurodegenerative disorder which eventually results iPalabras claves:Alzheimer disease, Amyloid hypotheses, Beta-amyloid, Beta-secretase, Gamma-secretaseAutores:Abad S., Antoni Camins, Ettcheto M., Folch J., Miguel Marin, Olloquequi J., Pedros I., Petrov D.Fuentes:scopusMasitinib for the treatment of mild to moderate Alzheimer's disease
ReviewAbstract: Alzheimer's disease (AD) is a degenerative neurological disorder that is the most common cause of dePalabras claves:ALZHÉIMER, inflammation, masitinib, Neurodegeneration, TauAutores:Abad S., Antoni Camins, Auladell C., Ettcheto M., Folch J., Lazarowski A., Miguel Marin, Olloquequi J., Pedros I., Petrov D., Zárate C.B.Fuentes:scopus