KCa3.1 transgene induction in murine intestinal epithelium causes duodenal chyme accumulation and impairs duodenal contractility

Article

Abstract: The epithelial intermediate-conductance calcium/calmodulin-regulated KCa3.1 channel is considered to

Palabras claves:

Contractility, Duodenum, Epithelium, Intermediate-conductance calcium-activated potassium channel, KCa3.1, Transgenic mice

Autores:

Calmarza P., Delgado-Wicke P., Esther Pueyo, Garcia-Otin A.L., Hamilton K.L., Köhler R., Lopez V., Lozano-Gerona J., Miura H., Oliván-Viguera A., Ramón-Gimenez M., Valero M.S., Valero S.

Fuentes:

scopus