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Decrease of calbindin-d28k, calretinin, and parvalbumin by taurine treatment does not induce a major susceptibility to kainic acid
ArticleAbstract: Taurine, 2-aminoethanesulfonic acid, is present at high concentrations in many invertebrate and vertPalabras claves:calcium, Epilepsy, neuronal death, neuroprotectionAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Porquet D., Romero R., Utrera J.Fuentes:scopusExpression pattern of ataxia telangiectasia mutated (ATM), p53, Akt, and glycogen synthase kinase-3β in the striatum of rats treated with 3-nitropropionic acid
ArticleAbstract: 3-Nitropropionic acid (3-NPA) is a mitochondrial toxin used in the laboratory to replicate neurodegePalabras claves:Immunohistochemistry, Neurotoxin, RATAutores:Antoni Camins, Del Valle J., Duran-Vilaregut J., Manich G., Pallás M., Pelegrí Gabalda C., Vilaplana J.Fuentes:scopusFlow cytometric study of mitochondrial dysfunction after AMPA receptor activation
ArticleAbstract: The effect of AMPA-receptor stimulation on MMP and on the concentration of intracellular calcium ([CPalabras claves:AMPA, Cerebellar granule cells, Flow cytometry, Intracellular calcium, Mitochondrial membrane potentialAutores:Aguirre L., Antoni Camins, Camarasa J., Escubedo E., Gabriel C., Pallás M., Sureda F.X.Fuentes:scopusNeuroprotective effects of caffeine against complex I inhibition-induced apoptosis are mediated by inhibition of the ATM/p53/E2F-1 path in cerebellar granule neurons
ArticleAbstract: The aim of the present study was to evaluate the neuroprotective effects of caffeine, an inhibitor oPalabras claves:Ataxia telangiectasia mutated, caffeine, Cerebellar granule cells, Complex I inhibition, E2F-1, MPP +Autores:Alvira D., Antoni Camins, Casadesús G., Folch J., Pallás M., Smith M.A., Yeste-Velasco M.Fuentes:scopusInhibitors of NO-synthase and donors of NO modulate kainic acid-induced damage in the rat hippocampus
ArticleAbstract: The effects of nitric oxide synthase (NOS) inhibitors, N(ω)-nitro-L-arginine and 7-nitroindazole, anPalabras claves:Excitotoxicity, Heat shock proteins, kainate, Nitric oxide, NO-synthase, [ H]PK 11195 3Autores:Antoni Camins, Camarasa J., Escubedo E., Friguls B., Gabriel C., Pallás M., Planas A.M., Sureda F.X.Fuentes:scopusPrevention of epilepsy by taurine treatments in mice experimental model
ArticleAbstract: An experimental model based on kainic acid (KA) injections replicates many phenomenological featuresPalabras claves:Kainic acid, Neurodegeneration, neuroprotectionAutores:Antoni Camins, Auladell C., Duque D., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopusTau hyperphosphorylation and axonal damage induced by N,N- diethyldithiocarbamate (DEDTC) treatment along late postnatal development is followed by a rescue during adulthood
ArticleAbstract: Axonal degeneration has been described as the pathological hallmark of peripheral neuropathies inducPalabras claves:Axonal damage, DEDTC, Postnatal development, Tau phosphorylationAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopusTaurine treatment inhibits CaMKII activity and modulates the presence of calbindin D28k, calretinin, and parvalbumin in the brain
ArticleAbstract: Taurine, 2-aminoethanesulfonic acid, is present at high concentrations in many invertebrate and vertPalabras claves:Calbindin D28k, Calretinin, CAMKII, Parvalbumin, taurineAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopus