Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice

Article

Abstract: Senescence-accelerated mice (SAMP8) and senescence-accelerated resistant mice (SAMR1) were studied a

Palabras claves:

aging, BRAIN, Melatonin receptor-1, Neurofibrillary tangle, Nuclear factor-kappa B, Retinoic acid receptor-related orphan receptor-α1, Senescence-accelerated mice

Autores:

Antoni Camins, Caballero B., Coto-Montes A., Gonzalo-Calvo D.D., Gutiérrez-Cuesta J., Huidobro-Fernández C., Pallás M., Rodríguez-Colunga M.J., Sierra V., Soria-Valles C., Tolivia D., Vega-Naredo I.

Fuentes:

scopus