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Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice
ArticleAbstract: Senescence-accelerated mice (SAMP8) and senescence-accelerated resistant mice (SAMR1) were studied aPalabras claves:aging, BRAIN, Melatonin receptor-1, Neurofibrillary tangle, Nuclear factor-kappa B, Retinoic acid receptor-related orphan receptor-α1, Senescence-accelerated miceAutores:Antoni Camins, Caballero B., Coto-Montes A., Gonzalo-Calvo D.D., Gutiérrez-Cuesta J., Huidobro-Fernández C., Pallás M., Rodríguez-Colunga M.J., Sierra V., Soria-Valles C., Tolivia D., Vega-Naredo I.Fuentes:scopusLoss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid
ArticleAbstract: MT1-MMP (MMP14) is a collagenolytic enzyme located at the cell surface and implicated in extracellulPalabras claves:aging, extracellular matrix, metalloprotease, Nuclear lamina, retinoidsAutores:Alina Aguirre, Fernández-García M.S., Fueyo A., Garabaya C., Gutiérrez-Abril J., Gutiérrez-Fernández A., Lõpez-Otín C., Osorio F.G., Puente X.S., Soria-Valles C.Fuentes:scopusMelatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated mice
ArticleAbstract: We studied the effect of age and melatonin on cell death processes in brain aging. Senescence-accelePalabras claves:aging, apoptosis, Autophagy, Calpains, Caspases, Cathepsins, melatoninAutores:Antoni Camins, Caballero B., Coto-Montes A., Gonzalo-Calvo D.D., Huidobro-Fernández C., Pallás M., Rodríguez-Colunga M.J., Sierra V., Soria-Valles C., Tolivia D., Vega-Naredo I.Fuentes:scopus