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Activation of the neuroprotective ERK signaling pathway by fructose-1,6-bisphosphate during hypoxia involves intracellular Ca<sup>2+</sup> and phospholipase C
ArticleAbstract: The mechanism of the neuroprotective action of the glycolytic pathway intermediate fructose-1,6-bispPalabras claves:6-Bisphosphate, Brain injury, CA1 neuron, calcium, Fructose-1, Hypoxia, ischemiaAutores:Bickler P.E., Fahlman C.S., George A. Gregory, Sullivan B.Fuentes:scopusFructose-1,6-bisphosphate after hypoxic ischemic injury is protective to the neonatal rat brain
ArticleAbstract: Fructose-1,6-bisphosphate (FBP) has been shown to attenuate central nervous system injury in adult aPalabras claves:Brain injury, Development, Hypocalcemia, Hypoxia, ischemia, neonate, therapyAutores:Berrios M., Ferriero D., George A. Gregory, Sheldon R., Sola A.Fuentes:scopusNeuroprotection and intracellular Ca<sup>2+</sup> modulation with fructose-1,6-bisphosphate during in vitro hypoxia-ischemia involves phospholipase C-dependent signaling
ArticleAbstract: The neuroprotectant fructose-1,6-bisphosphate (FBP) preserves cellular [ATP] and prevents catastrophPalabras claves:Brain injury, CA1 neuron, Cacium, Fructose-1,6-bishopsphate, Hypoxia, ischemiaAutores:Bickler P.E., Donohoe P., Fahlman C.S., George A. Gregory, Vexler Z.Fuentes:scopus