Mostrando 7 resultados de: 7
Filtros aplicados
Publisher
Age(1)
Cellular and Molecular Life Sciences(1)
International Journal of Developmental Neuroscience(1)
Journal of Alzheimer's Disease(1)
Journal of Neurochemistry(1)
Área temáticas
Fisiología humana(3)
Bioquímica(2)
Ciencias de la computación(1)
Farmacología y terapéutica(1)
Fisiología y materias afines(1)
Origen
scopus(7)
Cell cycle activation in striatal neurons from Huntington's disease patients and rats treated with 3-nitropropionic acid
ArticleAbstract: This study was undertaken to investigate the potential role of cell cycle re-entry in an experimentaPalabras claves:apoptosis, cell cycle, Huntington's disease, StriatumAutores:Antoni Camins, Crespo-Biel N., Del Valle J., Duran-Vilaregut J., Ferrer I., Pallás M., Pelegrí Gabalda C., Vilaplana J.Fuentes:scopusActivation of ataxia telangiectasia muted under experimental models and human Parkinson's disease
ArticleAbstract: In the present study we demonstrated that neurotoxin MPP +-induced DNA damage is followed by ataxiaPalabras claves:apoptosis, ATM, B65 neuroblastoma, cell cycle, MPP +, Parkinson's Disease, Retinoblastoma proteinAutores:Alvira D., Antoni Camins, de la Torre A.V., Ferrer I., Folch J., Gutiérrez-Cuesta J., Jordan J., Junyent F., Pallás M., Pizarro J.G., Sureda F.X., Verdaguer E.Fuentes:scopusActivation of the calpain/cdk5/p25 pathway in the girus cinguli in Parkinson's disease
ArticleAbstract: The mechanisms involved in neuronal loss in Parkinson's disease (PD) are not known, although recentPalabras claves:CDK5, cell cycle, E2F-1, Parkinson's DiseaseAutores:Alvira D., Antoni Camins, Ferrer I., Garcia-Castro B., Gutiérrez-Cuesta J., Pallás M.Fuentes:scopusAmyloid and tau pathology of familial Alzheimer’s disease APP/PS1 mouse model in a senescence phenotype background (SAMP8)
ArticleAbstract: The amyloid precursor protein/presenilin 1 (APP/PS1) mouse model of Alzheimer’s disease (AD) has proPalabras claves:aging, cognitive impairment, Familial Alzheimer’s disease (fAD), Tau hyperphosphorylation, β-AmyloidAutores:Andrés-Benito P., Antoni Camins, Canudas A.M., Del Valle J., Ferrer I., Griñan-Ferré C., Pallás M., Porquet D.Fuentes:scopusErratum: Differences in activation of ERK1/2 and p38 kinase in Jnk3 null mice following KA treatment (Journal of Neurochemistry (2010) 114 (1315-1322))
OtherAbstract:Palabras claves:Autores:Antoni Camins, Auladell C., De Lemos M.L., Ferrer I., Folch J., Junyent F., Pallás M., Romero R., Verdaguer E.Fuentes:scopusNeuroprotective role of trans-resveratrol in a murine model of familial alzheimer's disease
ArticleAbstract: The amyloid-β protein precursor/presenilin 1 (AβPP/PS1) mouse model of Alzheimer's disease (AD) hasPalabras claves:AMPK, inflammation, Mitochondria, Resveratrol, Sirtuin 1Autores:Antoni Camins, Del Valle J., Ferrer I., Griñan-Ferré C., Pallás M., Porquet D., Sanfeliu C.Fuentes:scopusModulation of SIRT1 expression in different neurodegenerative models and human pathologies
ArticleAbstract: We examined the expression of SIRT1 in several experimental paradigms of human pathologies. We usedPalabras claves:3-nitropropionic acid, BRAIN, cell cycle, kainate, MPP+, rotenoneAutores:Alvira D., Antoni Camins, Canudas A.M., Crespo-Biel N., Ferrer I., Folch J., Gutiérrez-Cuesta J., Pallás M., Pizarro J.G., Sureda F.X., Tajes M., Yeste-Velasco M.Fuentes:scopus