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Amyloid and tau pathology of familial Alzheimer’s disease APP/PS1 mouse model in a senescence phenotype background (SAMP8)
ArticleAbstract: The amyloid precursor protein/presenilin 1 (APP/PS1) mouse model of Alzheimer’s disease (AD) has proPalabras claves:aging, cognitive impairment, Familial Alzheimer’s disease (fAD), Tau hyperphosphorylation, β-AmyloidAutores:Andrés-Benito P., Antoni Camins, Canudas A.M., Del Valle J., Ferrer I., Griñan-Ferré C., Pallás M., Porquet D.Fuentes:scopusLithium treatment decreases activities of tau kinases in a murine model of senescence
ArticleAbstract: Lithium modulates glycogen synthase kinase 3β (GSK-3β), a kinase involved in Alzheimer disease-relatPalabras claves:aging, BRAIN, Lithium, Neurodegeneration, Senescence-accelerated mouseAutores:Antoni Camins, Caballero B., Casadesús G., Ferrer I., Folch J., Gutiérrez-Cuesta J., Pallás M., Smith M.A., Tajes M.Fuentes:scopus