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l-Isoaspartyl Methyltransferase Deficiency in Zebrafish Leads to Impaired Calcium Signaling in the Brain
ArticleAbstract: Isomerization of l-aspartyl and l-asparaginyl residues to l-isoaspartyl residues is one type of protPalabras claves:Calcium signaling, HT22 cells, isoaspartyl, protein repair, ZebrafishAutores:Clarke S.G., Conrotte J.F., Crawford A.D., Linster C.L., María Lorena Cordero-Maldonado, Martins T.G., Moein M., Skupin A., Soliman R., Warmack R.A.Fuentes:scopusNew insights into the early mechanisms of epileptogenesis in a zebrafish model of Dravet syndrome
ArticleAbstract: Objective: To pinpoint the earliest cellular defects underlying seizure onset (epileptogenic period)Palabras claves:Dravet syndrome, epileptogenesis, fenfluramine, Sodium channel, ZebrafishAutores:Brattespe J., Esguerra C.V., Gawel K., Grzyb K., Heintz S., Ju Xu, Kinga Gawel, María Lorena Cordero-Maldonado, Martina S., Poovathingal S.K., Sarah Heintz, Satheesh S.V., Skupin A., Suster M., Tiraboschi E., van der Ent W., Xu J.Fuentes:scopusSeizure-induced increase in microglial cell population in the developing zebrafish brain
ArticleAbstract: Epilepsy is a chronic brain disorder characterized by unprovoked and recurrent seizures, of which 60Palabras claves:Acute seizures, Developing brain, Epilepsy, kainate, Microglia, ZebrafishAutores:Corrado Ameli, Crawford A.D., Cristina Donato, Francesca Peri, Laurent Mombaerts, María Lorena Cordero-Maldonado, Martins T.G., Skupin A., Soliman R.Fuentes:scopus