Carbonyl stress and NMDA receptor activation contribute to methylglyoxal neurotoxicity

Article

Abstract: Methylglyoxal (MG) is a reactive α-ketoaldehyde physiologically generated as a by-product of glycoly

Palabras claves:

Carbonyl stress, Free radicals, methylglyoxal, Mitochondria, Neurodegeneration, NMDA receptors

Autores:

Allgaier C., Antoni Camins, Garcia De Arriba S., García-Jordà E., Krügel U., Lewerenz A., Münch G., Nieber K., Pallás M., Regenthal R., Verdaguer E., Vissiennon Z.

Fuentes:

scopus