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scopus(13)
A metabolic perspective of late onset Alzheimer's disease
ReviewAbstract: After decades of research, the molecular neuropathology of Alzheimer's disease (AD)is still one of tPalabras claves:Alzheimer's disease, c-Jun N-terminal kinase inhibitors, Insulin, Licochalcone A, neuroinflammation, Reticulum stress, Type 2 Diabetes mellitusAutores:Antoni Camins, Auladell C., Busquets O., Cano A., Castro-Torres R.D., Ettcheto M., Folch J., García M.L., Manzine P.R., Olloquequi J., Sanchez-Lopez E., Verdaguer E., Zárate C.B.Fuentes:scopusExcitotoxicity in the pathogenesis of neurological and psychiatric disorders: Therapeutic implications
ReviewAbstract: Neurological and psychiatric disorders are leading contributors to the global disease burden, havingPalabras claves:Alzheimer’s disease, Autism Spectrum Disorder, Depression, glutamate, strokeAutores:Antoni Camins, Auladell C., Binvignat O., Cornejo-Córdova E., Olloquequi J., Soriano F.X., Verdaguer E.Fuentes:scopusEarly Preclinical Changes in Hippocampal CREB-Binding Protein Expression in a Mouse Model of Familial Alzheimer’s Disease
ArticleAbstract: The molecular basis of memory loss in Alzheimer’s disease (AD), the main cause of senile dementia, iPalabras claves:Alzheimer disease, APPswe/PS1dE9, CBP, CREB, hippocampusAutores:Abad S., Antoni Camins, Auladell C., Busquets O., Carro E., Casadesús G., Ettcheto M., Folch J., Olloquequi J., Pallás M., Pedros I., Petrov D., Sanchez-Lopez E., Zárate C.B.Fuentes:scopusDual mkk4 and mkk7 gene deletion in adult mouse causes an impairment of hippocampal immature granule cells
ArticleAbstract: (1) Background: The c-Jun-NH2-terminal protein kinase (JNK) is a mitogen-activated protein kinase inPalabras claves:Cre-LoxP, DCX, hippocampus, MKK4, MKK7, PJNKAutores:Antoni Camins, Auladell C., Caruana P., Castro-Torres R.D., Ettcheto M., Leighton K.M., Olloquequi J., Steele L., Ureña J., Verdaguer E., Zárate C.B.Fuentes:scopusNeuroprotective Effects of the Absence of JNK1 or JNK3 Isoforms on Kainic Acid-Induced Temporal Lobe Epilepsy-Like Symptoms
ArticleAbstract: The activation of c-Jun-N-terminal kinases (JNK) pathway has been largely associated with the pathogPalabras claves:c-Jun N-terminal kinase, Excitotoxicity, hippocampus, inflammation, Kainic acid, Knockout mice, Neurodegeneration, neuroprotectionAutores:Antoni Camins, Auladell C., Castro-Torres R.D., De Lemos M.L., Folch J., Junyent F., Olloquequi J., Verdaguer E., Zárate C.B.Fuentes:scopusInvolvement of JNK1 in Neuronal Polarization During Brain Development
ReviewAbstract: The c-Jun N-terminal Kinases (JNKs) are a group of regulatory elements responsible for the control oPalabras claves:asymmetric division, DCX, JNK1, MAP1B, MIGRATION, NMDAR, SCG10, synaptogenesis, WDR62Autores:Antoni Camins, Auladell C., Busquets O., Castro-Torres R.D., Ettcheto M., Folch J., Olloquequi J., Parcerisas A., Verdaguer E., Zárate C.B.Fuentes:scopusJNK Isoforms Are Involved in the Control of Adult Hippocampal Neurogenesis in Mice, Both in Physiological Conditions and in an Experimental Model of Temporal Lobe Epilepsy
ArticleAbstract: Neurogenesis in the adult dentate gyrus (DG) of the hippocampus allows the continuous generation ofPalabras claves:adult hippocampal neurogenesis, JNK isoforms, Kainic acid, Knockout miceAutores:Antoni Camins, Auladell C., Busquets O., Castro-Torres R.D., Ettcheto M., Folch J., Landa J., Olloquequi J., Rabaza M., Verdaguer E., Zárate C.B.Fuentes:scopusJNK1 and JNK3: divergent functions in hippocampal metabolic-cognitive function
ArticleAbstract: Background and aim: The appearance of alterations in normal metabolic activity has been increasinglyPalabras claves:COGNITION, high-fat diet, JNK1, JNK3, metabolismAutores:Antoni Camins, Auladell C., Bulló M., Busquets O., Cantero J.L., Carro E., Casadesús G., Espinosa-Jimenez T., Ettcheto M., Folch J., Olloquequi J., Verdaguer E.Fuentes:scopusVulnerability of calbindin, calretinin and parvalbumin in a transgenic/knock-in APPswe/PS1dE9 mouse model of Alzheimer disease together with disruption of hippocampal neurogenesis
ArticleAbstract: The pathogenesis of Alzheimer disease (AD) is characterized by accumulation of β-amyloid protein inPalabras claves:Alzheimer disease, APPswe/PS1dE9 mouse, Calbindin, Calretinin, hippocampus, ParvalbuminAutores:Antoni Camins, Auladell C., Brox S., De Lemos M.L., Olloquequi J., Petrov D., Romero R., Verdaguer E.Fuentes:scopusc-Jun N-terminal Kinase 1 ablation protects against metabolic-induced hippocampal cognitive impairments
ArticleAbstract: Abstract: The development of metabolic alterations like insulin resistance has been associated withPalabras claves:c-Jun N-terminal Kinase 1, Cognitive impairments, high-fat dietAutores:Antoni Camins, Auladell C., Bulló M., Busquets O., Casadesús G., Castro-Torres R.D., Eritja À., Espinosa-Jimenez T., Ettcheto M., Folch J., Olloquequi J., Verdaguer E., Zárate C.B.Fuentes:scopus