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Erratum: Differences in activation of ERK1/2 and p38 kinase in Jnk3 null mice following KA treatment (Journal of Neurochemistry (2010) 114 (1315-1322))
OtherAbstract:Palabras claves:Autores:Antoni Camins, Auladell C., De Lemos M.L., Ferrer I., Folch J., Junyent F., Pallás M., Romero R., Verdaguer E.Fuentes:scopusVulnerability of calbindin, calretinin and parvalbumin in a transgenic/knock-in APPswe/PS1dE9 mouse model of Alzheimer disease together with disruption of hippocampal neurogenesis
ArticleAbstract: The pathogenesis of Alzheimer disease (AD) is characterized by accumulation of β-amyloid protein inPalabras claves:Alzheimer disease, APPswe/PS1dE9 mouse, Calbindin, Calretinin, hippocampus, ParvalbuminAutores:Antoni Camins, Auladell C., Brox S., De Lemos M.L., Olloquequi J., Petrov D., Romero R., Verdaguer E.Fuentes:scopusTau hyperphosphorylation and axonal damage induced by N,N- diethyldithiocarbamate (DEDTC) treatment along late postnatal development is followed by a rescue during adulthood
ArticleAbstract: Axonal degeneration has been described as the pathological hallmark of peripheral neuropathies inducPalabras claves:Axonal damage, DEDTC, Postnatal development, Tau phosphorylationAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopus