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Amyloid and tau pathology of familial Alzheimer’s disease APP/PS1 mouse model in a senescence phenotype background (SAMP8)
ArticleAbstract: The amyloid precursor protein/presenilin 1 (APP/PS1) mouse model of Alzheimer’s disease (AD) has proPalabras claves:aging, cognitive impairment, Familial Alzheimer’s disease (fAD), Tau hyperphosphorylation, β-AmyloidAutores:Andrés-Benito P., Antoni Camins, Canudas A.M., Del Valle J., Ferrer I., Griñan-Ferré C., Pallás M., Porquet D.Fuentes:scopusEnvironmental enrichment modified epigenetic mechanisms in SAMP8 mouse hippocampus by reducing oxidative stress and inflammaging and achieving neuroprotection
ArticleAbstract: With the increase in life expectancy, aging and age-related cognitive impairments are becoming one oPalabras claves:Environmental enrichment, Epigenetics, inflammation, Neurodegeneration, Oxidative StressAutores:Antoni Camins, Companys-Alemany J., Griñan-Ferré C., Ortuo-Sahagún D., Pallás M., Palomera-ávalos V., Pérez-Cáceres D., Puigoriol-Illamola D., Teresa Rodrigo M.Fuentes:scopusNeuroprotective role of trans-resveratrol in a murine model of familial alzheimer's disease
ArticleAbstract: The amyloid-β protein precursor/presenilin 1 (AβPP/PS1) mouse model of Alzheimer's disease (AD) hasPalabras claves:AMPK, inflammation, Mitochondria, Resveratrol, Sirtuin 1Autores:Antoni Camins, Del Valle J., Ferrer I., Griñan-Ferré C., Pallás M., Porquet D., Sanfeliu C.Fuentes:scopus