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P38 MAPK Inhibition Protects Against Glutamate Neurotoxicity and Modifies NMDA and AMPA Receptor Subunit Expression
ArticleAbstract: NMDA and AMPA receptors are thought to be responsible for Ca++ influx during glutamate-induced excitPalabras claves:Cell signaling, Excitotoxicity, Glu-R expression, glutamate, p38 MAPKAutores:Antoni Camins, Castañeda-Arellano R., Castro-Torres R.D., Gudiño-Cabrera G., Rivera-Cervantes M.C., y Velasco A.I.F., Zárate C.B.Fuentes:scopusImpact of New Drugs for Therapeutic Intervention in Alzheimer’s Disease
ReviewAbstract: The increases in population ageing and growth are leading to a boosting in the number of people liviPalabras claves:clinical studies, dementia, neuroinflammation, novel therapies, synapsesAutores:Antoni Camins, Auladell C., Cano A., Carrasco M., Espinosa T., Ettcheto M., Folch J., Gudiño-Cabrera G., Olloquequi J., Sanchez-Lopez E., Ureña-Guerrero M.E., Verdaguer E., Zárate C.B.Fuentes:scopusMicroarray analysis of rat hippocampus exposed to excitotoxicity: Reversal Na <sup>+</sup>/Ca <sup>2+</sup> exchanger NCX3 is overexpressed in glial cells
ArticleAbstract: Multiple factors are involved in the glutamate-induced excitotoxicity phenomenon, such as overload oPalabras claves:Arrays, Excitotoxicity, glutamate, hippocampus, NCX3Autores:Antoni Camins, Auladell C., Gudiño-Cabrera G., Junyent F., Ortuo-Sahagún D., Pallás M., Rivera-Cervantes M.C., Verdaguer E., Zárate C.B.Fuentes:scopus