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Comparative analysis of the effects of resveratrol in two apoptotic models: Inhibition of complex I and potassium deprivation in cerebellar neurons
ArticleAbstract: The mechanism involved in neuronal apoptosis is largely unknown. Studies performed on neuronal cellPalabras claves:Cerebellar granule cells, MPP+, Resveratrol, Serum and potassium withdrawal, Sirt1, sirtinolAutores:Alvira D., Antoni Camins, Canudas A.M., Folch J., Pallás M., Verdaguer E., Yeste-Velasco M.Fuentes:scopusAntiapoptotic effects of roscovitine on camptothecin-induced DNA damage in neuroblastoma cells
ArticleAbstract: In the present study dopaminergic neuroblastoma B65 cells were exposed to Camptothecin (CPT) (0.5-10Palabras claves:Ataxia telangiectasia mutated, camptothecin, cell cycle, RoscovitineAutores:Antoni Camins, Auladell C., Folch J., Junyent F., Pallás M., Pizarro J.G., Verdaguer E., Zárate C.B.Fuentes:scopusGSK3β inhibition is involved in the neuroprotective effects of cyclin-dependent kinase inhibitors in neurons
ArticleAbstract: In the present study, we evaluated the effects of roscovitine (Rosco) and flavopiridol (Flavo), bothPalabras claves:apoptosis, Cerebellar granular cells, flavopiridol, GSK3β, RoscovitineAutores:Antoni Camins, Auladell C., de la Torre A.V., Folch J., Junyent F., Pallás M., Pelegrí Gabalda C., Verdaguer E., Vilaplana J., Zárate C.B.Fuentes:scopusGene expression profile in JNK3 null mice: A novel specific activation of the PI3K/AKT pathway
ArticleAbstract: JNK3 is mainly expressed in the CNS and it plays a crucial role in neuronal death in several neurodePalabras claves:Akt, Gene profiling, JNK, Neurodegeneration, neuroprotection, PI3KAutores:Antoni Camins, Auladell C., De Lemos M.L., Ferrer I., Folch J., Junyent F., Ortuo-Sahagún D., Pallás M., Romero R., Verdaguer E., Zárate C.B.Fuentes:scopusNeuroprotection by c-Jun NH<inf>2</inf>-terminal kinase inhibitor SP600125 against potassium deprivation-induced apoptosis involves the Akt pathway and inhibition of cell cycle reentry
ArticleAbstract: Increasing evidence implicates the c-Jun NH2-terminal kinase (JNK) pathway in the regulation of apopPalabras claves:Akt, c-Jun NH2-terminal kinase, cell cycle, Cerebellar granule cells, SP600125Autores:Antoni Camins, Casadesús G., Folch J., Pallás M., Smith M.A., Yeste-Velasco M.Fuentes:scopusLack of Jun-N-terminal kinase 3 (JNK3) does not protect against neurodegeneration induced by 3-nitropropionic acid
ArticleAbstract: Aims: 3-Nitropropionic acid (3-NP) is a toxin that replicates most of the clinical and pathophysioloPalabras claves:Huntington, JNK3, Neurodegeneration, Nitropropionic acidAutores:Antoni Camins, Auladell C., De Lemos M.L., Folch J., Junyent F., Pallás M., Verdaguer E., Zárate C.B.Fuentes:scopusOxidative stress-induced DNA damage and cell cycle regulation in B65 dopaminergic cell line
ArticleAbstract: Reactive oxygen species and oxidative stress are associated with neuronal cell death in many neurodePalabras claves:apoptosis, ATM, cell cycle, KU-55933, Retinoblastoma proteinAutores:Antoni Camins, Folch J., Jordan J., Junyent F., Pallás M., Pizarro J.G., Vázquez De La Torre A., Verdaguer E.Fuentes:scopusInhibition of multiple pathways accounts for the antiapoptotic effects of flavopiridol on potassium withdrawal-induced apoptosis in neurons
ArticleAbstract: Serum and potassium (S/K) deprivation is a well-known apoptotic model in cerebellar granule neuronsPalabras claves:apoptosis, Cdk, cell cycle, flavopiridol, GSK-3β, MEF2Autores:Alvira D., Antoni Camins, Canudas A.M., Folch J., Jiménez A., Jordá E.G., Pallás M., Rimbau V., Verdaguer E.Fuentes:scopusMelatonin suppresses nitric oxide production in glial cultures by pro-inflammatory cytokines through p38 MAPK inhibition
ArticleAbstract: Melatonin has been shown to down-regulate inflammatory responses and provide neuroprotection. HowevePalabras claves:Glial cells, inflammation, melatonin, Nitric oxideAutores:Antoni Camins, Auladell C., De Lemos M.L., Folch J., Junyent F., Martínez N., Pallás M., Patraca I., Verdaguer E., Vilar A.Fuentes:scopusModulation of SIRT1 expression in different neurodegenerative models and human pathologies
ArticleAbstract: We examined the expression of SIRT1 in several experimental paradigms of human pathologies. We usedPalabras claves:3-nitropropionic acid, BRAIN, cell cycle, kainate, MPP+, rotenoneAutores:Alvira D., Antoni Camins, Canudas A.M., Crespo-Biel N., Ferrer I., Folch J., Gutiérrez-Cuesta J., Pallás M., Pizarro J.G., Sureda F.X., Tajes M., Yeste-Velasco M.Fuentes:scopus