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Carbonyl stress and NMDA receptor activation contribute to methylglyoxal neurotoxicity
ArticleAbstract: Methylglyoxal (MG) is a reactive α-ketoaldehyde physiologically generated as a by-product of glycolyPalabras claves:Carbonyl stress, Free radicals, methylglyoxal, Mitochondria, Neurodegeneration, NMDA receptorsAutores:Allgaier C., Antoni Camins, Garcia De Arriba S., García-Jordà E., Krügel U., Lewerenz A., Münch G., Nieber K., Pallás M., Regenthal R., Verdaguer E., Vissiennon Z.Fuentes:scopusKainic acid-induced apoptosis in cerebellar granule neurons: An attempt at cell cycle re-entry
ArticleAbstract: This study was undertaken to investigate whether kainic acid (KA) may regulate the expression of sevPalabras claves:apoptosis, Cdk, cell cycle, Cyclines, E2F-1, Kainic acidAutores:Antoni Camins, Canudas A.M., Domínguez E., Escubedo E., García-Jordà E., Jiménez A., Pallás M., Pubill D., Verdaguer E.Fuentes:scopusKainic acid-induced neuronal cell death in cerebellar granule cells is not prevented by caspase inhibitors
ArticleAbstract: 1. We examined the role of non-NMDA receptors in kainic acid (KA)-induced apoptosis in cultures of rPalabras claves:Ac-DEVD-CHO, apoptosis, Calpain, Caspases, Cerebellar granule cells, colchicine, glutamate, Kainic acid, Par-4, Z-VAD.fmkAutores:Antoni Camins, Camarasa J., Canudas A.M., Escubedo E., García-Jordà E., Jiménez A., Pallás M., Stranges A., Sureda F.X., Verdaguer E.Fuentes:scopus