Neuroprotection by c-Jun NH<inf>2</inf>-terminal kinase inhibitor SP600125 against potassium deprivation-induced apoptosis involves the Akt pathway and inhibition of cell cycle reentry

Article

Abstract: Increasing evidence implicates the c-Jun NH2-terminal kinase (JNK) pathway in the regulation of apop

Palabras claves:

Akt, c-Jun NH2-terminal kinase, cell cycle, Cerebellar granule cells, SP600125

Autores:

Antoni Camins, Casadesús G., Folch J., Pallás M., Smith M.A., Yeste-Velasco M.

Fuentes:

scopus