Castro-Torres R.D.
201
Coauthors
20
Documentos
Volumen de publicaciones por año
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Año de publicación | Num. Publicaciones |
---|---|
2014 | 1 |
2015 | 2 |
2018 | 5 |
2019 | 5 |
2020 | 4 |
2021 | 3 |
Publicaciones por áreas de conocimiento
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Área de conocimiento | Num. Publicaciones |
---|---|
Neuropsicología | 13 |
Neurología | 11 |
Biología molecular | 4 |
Metabolismo | 2 |
Farmacología | 2 |
Medicamento | 2 |
Nutrición | 2 |
Biología del desarrollo | 2 |
Diabetes | 2 |
Medicina interna | 1 |
Publicaciones por áreas temáticas
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Área temática | Num. Publicaciones |
---|---|
Enfermedades | 18 |
Fisiología humana | 14 |
Farmacología y terapéutica | 6 |
Sistemas fisiológicos específicos de los animales | 3 |
Ginecología, obstetricia, pediatría, geriatría | 2 |
Mammalia | 1 |
Medicina y salud | 1 |
Bioquímica | 1 |
Microorganismos, hongos y algas | 1 |
Fisiología y materias afines | 1 |
Principales fuentes de datos
Origen | Num. Publicaciones |
---|---|
Scopus | 20 |
Google Scholar | 0 |
RRAAE | 0 |
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Coautores destacados por número de publicaciones
Coautor | Num. Publicaciones |
---|---|
Antoni Camins | 20 |
Zárate C.B. | 17 |
Auladell C. | 17 |
Verdaguer E. | 16 |
Ettcheto M. | 16 |
Busquets O. | 15 |
Folch J. | 15 |
Olloquequi J. | 14 |
Manzine P.R. | 8 |
Sanchez-Lopez E. | 6 |
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Publicaciones del autor
P38 MAPK Inhibition Protects Against Glutamate Neurotoxicity and Modifies NMDA and AMPA Receptor Subunit Expression
ArticleAbstract: NMDA and AMPA receptors are thought to be responsible for Ca++ influx during glutamate-induced excitPalabras claves:Cell signaling, Excitotoxicity, Glu-R expression, glutamate, p38 MAPKAutores:Antoni Camins, Castañeda-Arellano R., Castro-Torres R.D., Gudiño-Cabrera G., Rivera-Cervantes M.C., y Velasco A.I.F., Zárate C.B.Fuentes:scopusC-Jun N-Terminal Kinases in Alzheimer's Disease: A Possible Target for the Modulation of the Earliest Alterations
ReviewAbstract: Given the highly multifactorial origin of Alzheimer's disease (AD) neuropathology, disentangling andPalabras claves:Alzheimer's disease, Amyloid-β, axonal transport, hyperphosphorylated tau, JNK, synapse lossAutores:Antoni Camins, Auladell C., Busquets O., Castro-Torres R.D., Ettcheto M., Parcerisas A., Verdaguer E., Zárate C.B.Fuentes:scopusA single dose of pirfenidone attenuates neuronal loss and reduces lipid peroxidation after kainic acid-induced excitotoxicity in the pubescent rat hippocampus
ArticleAbstract: Systemic administration of kainic acid (KA) in rodents triggers limbic seizures following selectivePalabras claves:4-Hydroxynonenal (4-HNE), Kainic Acid Model, Neurodegeneration, neuroprotection, PirfenidoneAutores:Antoni Camins, Armendáriz-Borunda J., Bañuelos-Pineda J., Castro-Torres R.D., Chaparro-Huerta V., Flores-Soto M.E., Orozco-Suárez S.A., Zárate C.B.Fuentes:scopusPirfenidone Attenuates Microglial Reactivity and Reduces Inducible Nitric Oxide Synthase mRNA Expression After Kainic Acid-Mediated Excitotoxicity in Pubescent Rat Hippocampus
ArticleAbstract: Excitotoxicity and neuroinflammation are strongly linked to the progressions of neurodegenerative diPalabras claves:cytokines, iNOS, Kainic acid, Microglial reactivity, PirfenidoneAutores:Antoni Camins, Armendáriz-Borunda J., Castro-Torres R.D., Chaparro-Huerta V., Flores-Soto M.E., Jave-Suárez L., Mena-Munguía S., Zárate C.B.Fuentes:scopusJNK1 inhibition by Licochalcone A leads to neuronal protection against excitotoxic insults derived of kainic acid
ArticleAbstract: The mitogen-activated protein kinase family (MAPK) is an important group of enzymes involved in cellPalabras claves:Epilepsy, hippocampus, Kainic acid, Licochalcone A, NeurodegenerationAutores:Antoni Camins, Auladell C., Busquets O., Castro-Torres R.D., Ettcheto M., Folch J., Verdaguer E., Zárate C.B.Fuentes:scopus