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Chronic exposure to methylphenidate impairs appendicular bone quality in young rats
ArticleAbstract: Methylphenidate (MP) is a psychostimulant widely prescribed to treat Attention Deficit HyperactivityPalabras claves:Biomechanics, DXA, Growth suppression, methylphenidate, TestosteroneAutores:Ananth M., David Edward Komatsu, Hadjiargyrou M., Janda H.A., John C.M., Mary M.N., Robison L.S., Swanson J.M., Thanos P.K., Volkow N.D.Fuentes:scopusChronic oral methylphenidate treatment in adolescent rats promotes dose-dependent effects on NMDA receptor binding
ArticleAbstract: Aim: Examine the effects of chronic oral Methylphenidate (MP) treatment on the N-Methyl-D-aspartic aPalabras claves:ADOLESCENCE, methylphenidate, NMDA, ritalinAutores:David Edward Komatsu, Delis F., Hadjiargyrou M., Hamilton J., Jalloh K., Roeder N., Thanos P.K.Fuentes:scopusChronic oral methylphenidate treatment reversibly increases striatal dopamine transporter and dopamine type 1 receptor binding in rats
ArticleAbstract: Previously, we created an 8-h limited-access dual bottle drinking paradigm to deliver methylphenidatPalabras claves:dopamine, methylphenidate, Psychostimulant, Reward deficiency syndrome, reward, addiction, TransporterAutores:Ananth M., David Edward Komatsu, Hadjiargyrou M., Robison L.S., Thanos P.K.Fuentes:scopusChronic treatment and abstinence from methylphenidate exposure dose-dependently changes glucose metabolism in the rat brain
ArticleAbstract: Methylphenidate (MP) is extensively prescribed for attention deficit hyperactivity disorder (ADHD).Palabras claves:Brain Glucose Metabolism, FDG, imaging, methylphenidate, PetAutores:Blum K., David Edward Komatsu, Delis F., Fricke D., Hadjiargyrou M., Hamilton J., Martin C., Richer K., Sajjad M., Thanos P.K., Yao R.Fuentes:scopusCombined methylphenidate and fluoxetine treatment in adolescent rats significantly impairs weight gain with minimal effects on skeletal development
ArticleAbstract: Methylphenidate (MP) is frequently prescribed to treat Attention-Deficit/Hyperactivity Disorder (ADHPalabras claves:Fluoxetine, methylphenidate, RAT, Skeletal developmentAutores:Areikat N., Chirokikh A.A., Connor C., David Edward Komatsu, Duque E., Hadjiargyrou M., Jones R., Thanos P.K., Uddin S.M.Z.Fuentes:scopusBehavioral, Neurochemical and Developmental Effects of Chronic Oral Methylphenidate: A Review
ReviewAbstract: The majority of animal studies on methylphenidate (MP) use intraperitoneal (IP) injections, subcutanPalabras claves:ADHD, animal model, Behavior, dopamine, methylphenidate, neurochemistry, Psychostimulant, ritalinAutores:Ahmed R., Arnavut E., Badgaiyan R.D., Blum K., Carvalho A., David Edward Komatsu, Hadjiargyrou M., Lee W.X., Senior D., Thanos P.K.Fuentes:scopusDelivery of rhBMP-2 plasmid DNA complexes via a PLLA/collagen electrospun scaffold induces ectopic bone formation
ArticleAbstract: The development of effective strategies for gene delivery is a critical goal in DNA-based tissue engPalabras claves:BMP-2, Bone formation, complexes, Electrospun, scaffoldAutores:David Edward Komatsu, Hadjiargyrou M., Zhao X.Fuentes:scopusAbstinence following intermittent methylphenidate exposure dose-dependently modifies brain glucose metabolism in the rat brain
ArticleAbstract: Methylphenidate (MP) is a psychostimulant chronically prescribed for the treatment of attention defiPalabras claves:Abstinence, addiction, brain function, FDG, Functional connectivity, glucose utilization, methylphenidate, microPET, PetAutores:Arnavut E., David Edward Komatsu, Hadjiargyrou M., Hamilton J., Sajjad M., Thanos P.K., Yao R.Fuentes:scopusActivation of the transcription factor HIF-1 and its target genes, VEGF, HO-1, iNOS, during fracture repair
ArticleAbstract: One of the immediate sequelae of bone fracture is regional hypoxia resulting from vasculature disrupPalabras claves:Angiogenesis, bone, Callus, Fracture, HIF-1α, HypoxiaAutores:David Edward Komatsu, Hadjiargyrou M.Fuentes:scopusEnhanced bone regeneration associated with decreased apoptosis in mice with partial HIF-1α deficiency
ArticleAbstract: HIF-1α activates genes under hypoxia and was hypothesized to regulate bone regeneration. SurprisinglPalabras claves:apoptosis, bone, Fracture, Hypoxia-inducible factor 1α, REGENERATIONAutores:Bosch-Marce M., David Edward Komatsu, Hadjiargyrou M., Semenza G.L.Fuentes:scopus