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Molecular Neurobiology(3)
Journal of Alzheimer's Disease(2)
Biochimica et Biophysica Acta - Molecular Basis of Disease(1)
Experimental Gerontology(1)
Frontiers in Bioscience - Landmark(1)
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scopus(13)
Dexibuprofen prevents neurodegeneration and cognitive decline in APPswe/PS1dE9 through multiple signaling pathways
ArticleAbstract: The aim of the present study is to elucidate the neuronal pathways associated to NSAIDs causing a rePalabras claves:Alzheimer's disease, APPswe/PS1dE9, Dexibuprofen, hippocampus, insulin receptor, Memory impairment, Mitochondria, TauAutores:Antoni Camins, Auladell C., Busquets O., Ettcheto M., Folch J., García M.L., Olloquequi J., Pallás M., Pons L., Sanchez-Lopez E., Zárate C.B.Fuentes:scopusGSPE pre-treatment protects against long-term cafeteria diet-induced mitochondrial and inflammatory affectations in the hippocampus of rats
ArticleAbstract: Deregulations like the loss of sensitivity to insulin (insulin resistance) and chronic inflammationPalabras claves:BRAIN, Cafeteria, diet, GSPE, hippocampus, inflammation, metabolism, StressAutores:Antoni Camins, Auladell C., Bulló M., Busquets O., Carrasco M., Espinosa-Jimenez T., Ettcheto M., Folch J., Pinent M., Rodríguez-Gallego E., Verdaguer E.Fuentes:scopusEpigallocatechin-3-Gallate (EGCG) Improves Cognitive Deficits Aggravated by an Obesogenic Diet Through Modulation of Unfolded Protein Response in APPswe/PS1dE9 Mice
ArticleAbstract: Epigallocatechin-3-gallate (EGCG), a catechin found in green tea, has been previously investigated fPalabras claves:APPswe/PS1dE9 mice, Cognitive deficits, Epigallocatechin-3-gallate, hippocampus, obesity, unfolded protein responseAutores:Antoni Camins, Auladell C., Busquets O., Cano A., Castro-Torres R.D., Ettcheto M., Folch J., García M.L., Manzine P.R., Olloquequi J., Verdaguer E., Zárate C.B.Fuentes:scopusEvaluation of neuropathological effects of a high-fat diet in a presymptomatic Alzheimer's disease stage in APP/PS1 mice
ArticleAbstract: Alzheimer's disease (AD) is currently an incurable aging-related neurodegenerative disorder. RecentPalabras claves:Alzheimer's disease, APPswe/PS1dE9, hippocampus, insulin receptor, Mitochondria, TauAutores:Alva N., Antoni Camins, Auladell C., Carbonell T., Ettcheto M., Folch J., Pallás M., Pedros I., Petrov D., Zárate C.B.Fuentes:scopusEarly Preclinical Changes in Hippocampal CREB-Binding Protein Expression in a Mouse Model of Familial Alzheimer’s Disease
ArticleAbstract: The molecular basis of memory loss in Alzheimer’s disease (AD), the main cause of senile dementia, iPalabras claves:Alzheimer disease, APPswe/PS1dE9, CBP, CREB, hippocampusAutores:Abad S., Antoni Camins, Auladell C., Busquets O., Carro E., Casadesús G., Ettcheto M., Folch J., Olloquequi J., Pallás M., Pedros I., Petrov D., Sanchez-Lopez E., Zárate C.B.Fuentes:scopusEarly alterations in energy metabolism in the hippocampus of APPswe/PS1dE9 mouse model of Alzheimer's disease
ArticleAbstract: The present study had focused on the behavioral phenotype and gene expression profile of molecules rPalabras claves:Alzheimer's disease, APPswe/PS1dE9, hippocampus, insulin receptor, Mitochondria, TauAutores:Allgaier M., Antoni Camins, Auladell C., Barroso E., Casadesús G., Folch J., Pallás M., Pedros I., Petrov D., Sureda F.X., Vázquez-Carrera M., Zárate C.B.Fuentes:scopusDual mkk4 and mkk7 gene deletion in adult mouse causes an impairment of hippocampal immature granule cells
ArticleAbstract: (1) Background: The c-Jun-NH2-terminal protein kinase (JNK) is a mitogen-activated protein kinase inPalabras claves:Cre-LoxP, DCX, hippocampus, MKK4, MKK7, PJNKAutores:Antoni Camins, Auladell C., Caruana P., Castro-Torres R.D., Ettcheto M., Leighton K.M., Olloquequi J., Steele L., Ureña J., Verdaguer E., Zárate C.B.Fuentes:scopusNeuroprotective Effects of the Absence of JNK1 or JNK3 Isoforms on Kainic Acid-Induced Temporal Lobe Epilepsy-Like Symptoms
ArticleAbstract: The activation of c-Jun-N-terminal kinases (JNK) pathway has been largely associated with the pathogPalabras claves:c-Jun N-terminal kinase, Excitotoxicity, hippocampus, inflammation, Kainic acid, Knockout mice, Neurodegeneration, neuroprotectionAutores:Antoni Camins, Auladell C., Castro-Torres R.D., De Lemos M.L., Folch J., Junyent F., Olloquequi J., Verdaguer E., Zárate C.B.Fuentes:scopusMicroarray analysis of rat hippocampus exposed to excitotoxicity: Reversal Na <sup>+</sup>/Ca <sup>2+</sup> exchanger NCX3 is overexpressed in glial cells
ArticleAbstract: Multiple factors are involved in the glutamate-induced excitotoxicity phenomenon, such as overload oPalabras claves:Arrays, Excitotoxicity, glutamate, hippocampus, NCX3Autores:Antoni Camins, Auladell C., Gudiño-Cabrera G., Junyent F., Ortuo-Sahagún D., Pallás M., Rivera-Cervantes M.C., Verdaguer E., Zárate C.B.Fuentes:scopusJNK1 inhibition by Licochalcone A leads to neuronal protection against excitotoxic insults derived of kainic acid
ArticleAbstract: The mitogen-activated protein kinase family (MAPK) is an important group of enzymes involved in cellPalabras claves:Epilepsy, hippocampus, Kainic acid, Licochalcone A, NeurodegenerationAutores:Antoni Camins, Auladell C., Busquets O., Castro-Torres R.D., Ettcheto M., Folch J., Verdaguer E., Zárate C.B.Fuentes:scopus