Pedros I.
131
Coauthors
14
Documentos
Volumen de publicaciones por año
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Año de publicación | Num. Publicaciones |
---|---|
2012 | 1 |
2013 | 1 |
2014 | 2 |
2015 | 5 |
2016 | 2 |
2017 | 1 |
2018 | 2 |
Publicaciones por áreas de conocimiento
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Área de conocimiento | Num. Publicaciones |
---|---|
Neurología | 11 |
Neuropsicología | 7 |
Medicamento | 4 |
Metabolismo | 2 |
Inmunología | 1 |
Inflamación | 1 |
Biología molecular | 1 |
Medicina interna | 1 |
Publicaciones por áreas temáticas
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Área temática | Num. Publicaciones |
---|---|
Enfermedades | 12 |
Fisiología humana | 10 |
Farmacología y terapéutica | 5 |
Problemas sociales y servicios a grupos | 3 |
Salud y seguridad personal | 1 |
Sistemas fisiológicos específicos de los animales | 1 |
Principales fuentes de datos
Origen | Num. Publicaciones |
---|---|
Scopus | 14 |
Google Scholar | 0 |
RRAAE | 0 |
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Coautores destacados por número de publicaciones
Coautor | Num. Publicaciones |
---|---|
Antoni Camins | 14 |
Folch J. | 14 |
Petrov D. | 11 |
Zárate C.B. | 10 |
Pallás M. | 9 |
Ettcheto M. | 8 |
Auladell C. | 8 |
Abad S. | 6 |
Sureda F.X. | 6 |
Miguel Marin | 4 |
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Top Keywords
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Publicaciones del autor
Metabolic basis of sporadic Alzeimer’s disease. Role of hormones related to energy metabolism
ArticleAbstract: The more common sporadic form of Alzheimer disease (SAD) and the metabolic syndrome are two highly pPalabras claves:Adipokines, amylin, Ghrelin, leptin, prolactin, Sporadic Alzheimer diseaseAutores:Antoni Camins, Folch J., Martínez N., Patraca I., Pedros I., Sureda F.X.Fuentes:scopusReview of the advances in treatment for Alzheimer disease: Strategies for combating β-amyloid protein
OtherAbstract: Introduction Alzheimer disease (AD) is a major neurodegenerative disorder which eventually results iPalabras claves:Alzheimer disease, Amyloid hypotheses, Beta-amyloid, Beta-secretase, Gamma-secretaseAutores:Abad S., Antoni Camins, Ettcheto M., Folch J., Miguel Marin, Olloquequi J., Pedros I., Petrov D.Fuentes:scopusHypercholesterolemia and neurodegeneration. Comparison of hippocampal phenotypes in LDLr knockout and APPswe/PS1dE9 mice
ArticleAbstract: Previous studies suggest that Alzheimer's disease (AD) neurobiology could not be explained solely byPalabras claves:Alzheimer's disease, APP/PS1, cholesterol, hippocampus, LDLR-/-, OXPHOSAutores:Alegret M., Antoni Camins, De Lemos M.L., Ettcheto M., Folch J., Laguna J.C., Pallás M., Pedros I., Petrov D.Fuentes:scopusAdipokine pathways are altered in hippocampus of an experimental mouse model of Alzheimer’s disease
ArticleAbstract: A growing body of evidence suggests that β-amyloid peptides (Aβ) are unlikely to be the only factorPalabras claves:ALZHÉIMER, APP/PS1, hippocampus, leptin, prolactinAutores:Abad S., Antoni Camins, Artiach G., Folch J., Pallás M., Pedros I., Petrov D., Ramon-Duaso C., Sureda F.X., Zárate C.B.Fuentes:scopusEvaluation of neuropathological effects of a high-fat diet in a presymptomatic Alzheimer's disease stage in APP/PS1 mice
ArticleAbstract: Alzheimer's disease (AD) is currently an incurable aging-related neurodegenerative disorder. RecentPalabras claves:Alzheimer's disease, APPswe/PS1dE9, hippocampus, insulin receptor, Mitochondria, TauAutores:Alva N., Antoni Camins, Auladell C., Carbonell T., Ettcheto M., Folch J., Pallás M., Pedros I., Petrov D., Zárate C.B.Fuentes:scopus