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Decrease of calbindin-d28k, calretinin, and parvalbumin by taurine treatment does not induce a major susceptibility to kainic acid
ArticleAbstract: Taurine, 2-aminoethanesulfonic acid, is present at high concentrations in many invertebrate and vertPalabras claves:calcium, Epilepsy, neuronal death, neuroprotectionAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Porquet D., Romero R., Utrera J.Fuentes:scopusTau hyperphosphorylation and axonal damage induced by N,N- diethyldithiocarbamate (DEDTC) treatment along late postnatal development is followed by a rescue during adulthood
ArticleAbstract: Axonal degeneration has been described as the pathological hallmark of peripheral neuropathies inducPalabras claves:Axonal damage, DEDTC, Postnatal development, Tau phosphorylationAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopusTaurine treatment inhibits CaMKII activity and modulates the presence of calbindin D28k, calretinin, and parvalbumin in the brain
ArticleAbstract: Taurine, 2-aminoethanesulfonic acid, is present at high concentrations in many invertebrate and vertPalabras claves:Calbindin D28k, Calretinin, CAMKII, Parvalbumin, taurineAutores:Antoni Camins, Auladell C., De Lemos M.L., Junyent F., Pallás M., Romero R., Utrera J.Fuentes:scopus