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Coexistence of accident aneurismatic subarachnoid hemorrhage and pulmonary thromboembolic in a patient with COVID-19. Report of a case

Abstract:

COVID-19 is caused by SARS-CoV2, a beta coronavirus currently responsible for a pandemic with a high rate of contagiousness and mortality worldwide. In addition to its impact at the lung level, this virus can trigger a storm of proinflammatory cytokines that damage the vascular endothelium, generate endothelium, endothelial dysfunction, and a high prevalence of thrombotic events. Clinically, this phenomenon's expression is evidenced by high D-dimer levels, venous and arterial thrombosis, pulmonary embolism, cerebrovascular and cardiovascular diseases, and diffuse microvascular thrombosis. Since the endothelium is a vital organ in regulating vascular tone and homeostasis, its involvement leads to vasoconstriction, ischemia, infarction, and inflammation, which possibly causes a weakening of its structure. In the following case report, we narrate the history of a patient with SARS-CoV2 pneumonia, who presented concomitance of hemorrhagic and thrombotic endothelial alterations, expressed by an accidental aneurysmal subarachnoid hemorrhage and submassive pulmonary thromboembolism.