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HEREDITARY SENSORY AND AUTONOMIC NEUROPATHY CHARACTERIZED BY HYPERTROPHIC NEUROPATHY, GASTROINTESTINAL DYSAUTONOMIA, NERVE DEAFNESS, AND OPHTHALMOPLEGIA-A NEW TYPE
OtherAbstract:Palabras claves:Autores:Miguel Angel Herrera CangoFuentes:googleHigh-fat diet-induced deregulation of hippocampal insulin signaling and mitochondrial homeostasis deficiences contribute to Alzheimer disease pathology in rodents
ArticleAbstract: Global obesity is a pandemic status, estimated to affect over 2 billion people, that has resulted inPalabras claves:Alzheimer disease, APPswe/PS1dE9, hippocampus, insulin receptor, Mitochondria, TauAutores:Antoni Camins, Artiach G., Barroso E., Carro E., Casadesús G., Ferrer I., Folch J., Pallás M., Pedros I., Petrov D., Sureda F.X., Vázquez-Carrera M., Zárate C.B.Fuentes:scopusHypercholesterolemia and neurodegeneration. Comparison of hippocampal phenotypes in LDLr knockout and APPswe/PS1dE9 mice
ArticleAbstract: Previous studies suggest that Alzheimer's disease (AD) neurobiology could not be explained solely byPalabras claves:Alzheimer's disease, APP/PS1, cholesterol, hippocampus, LDLR-/-, OXPHOSAutores:Alegret M., Antoni Camins, De Lemos M.L., Ettcheto M., Folch J., Laguna J.C., Pallás M., Pedros I., Petrov D.Fuentes:scopusElevated oxidative stress in the brain of senescence-accelerated mice at 5 months of age
ArticleAbstract: The senescence-accelerated mouse (SAM) is a useful animal model to study aging or age-associated disPalabras claves:aging, Alpha-synuclein, Antioxidant defence, Brain damage, Gliosis, Oxidative Stress, Senescence-accelerated model mouse (SAM), Tau phosphorylationAutores:Álvarez-García O., Antoni Camins, Caballero B., Coto-Montes A., García J., Pallás M., Sierra V., Tomás-Zapico C., Vega-Naredo I.Fuentes:scopusEnvironmental enrichment modified epigenetic mechanisms in SAMP8 mouse hippocampus by reducing oxidative stress and inflammaging and achieving neuroprotection
ArticleAbstract: With the increase in life expectancy, aging and age-related cognitive impairments are becoming one oPalabras claves:Environmental enrichment, Epigenetics, inflammation, Neurodegeneration, Oxidative StressAutores:Antoni Camins, Companys-Alemany J., Griñan-Ferré C., Ortuo-Sahagún D., Pallás M., Palomera-ávalos V., Pérez-Cáceres D., Puigoriol-Illamola D., Teresa Rodrigo M.Fuentes:scopusEpigallocatechin-3-gallate loaded PEGylated-PLGA nanoparticles: A new anti-seizure strategy for temporal lobe epilepsy
ArticleAbstract: Temporal lobe epilepsy is the most common type of pharmacoresistant epilepsy in adults. EpigallocatePalabras claves:Epigallocatechin-3-gallate, Epilepsy, PLGA-PEG, Polymeric nanoparticlesAutores:Antoni Camins, Auladell C., Barenys M., Calpena A.C., Cano A., Espina M., Ettcheto M., Folch J., García M.L., Sanchez-Lopez E.Fuentes:scopusErratum: Cerebral amyloid angiopathy, blood-brain barrier disruption and amyloid accumulation in SAMP8 mice. (Neurodegenerative Diseases (2011) 8 (421-429) DOI:10.1159/000324757)
OtherAbstract:Palabras claves:Autores:Antoni Camins, Del Valle J., Duran-Vilaregut J., Manich G., Pallás M., Pelegr C., Vilaplana J.Fuentes:scopusErratum: Differences in activation of ERK1/2 and p38 kinase in Jnk3 null mice following KA treatment (Journal of Neurochemistry (2010) 114 (1315-1322))
OtherAbstract:Palabras claves:Autores:Antoni Camins, Auladell C., De Lemos M.L., Ferrer I., Folch J., Junyent F., Pallás M., Romero R., Verdaguer E.Fuentes:scopusExtracellular vesicles, the emerging mirrors of brain physiopathology
ArticleAbstract: Extracellular vesicles are secreted by a wide variety of cells, and their primary functions includePalabras claves:Alzheimer’s disease, exosomes, Extracellular vesicles, Glioblastoma, multiple sclerosis, amyotrophic lateral sclerosis, neurodegenerative diseases, Parkinson’s DiseaseAutores:Antoni Camins, Bernuz M., Boada M., Cano A., de Antonio E.E., Ettcheto M., Martí M., Pividori M.I., Puerta R., Ruíz A., Sanchez-Lopez E., Souto E.M.B.Fuentes:scopusFavorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice
ArticleAbstract: Senescence-accelerated mice (SAMP8) and senescence-accelerated resistant mice (SAMR1) were studied aPalabras claves:aging, BRAIN, Melatonin receptor-1, Neurofibrillary tangle, Nuclear factor-kappa B, Retinoic acid receptor-related orphan receptor-α1, Senescence-accelerated miceAutores:Antoni Camins, Caballero B., Coto-Montes A., Gonzalo-Calvo D.D., Gutiérrez-Cuesta J., Huidobro-Fernández C., Pallás M., Rodríguez-Colunga M.J., Sierra V., Soria-Valles C., Tolivia D., Vega-Naredo I.Fuentes:scopus